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《联合生物学医学期刊》

　

《联合生物学医学期刊》2007年第03期

three-dimensional em structure of the ectodomain of integrin v3 in a complex with fibronectin
the aaa+ protein torsina interacts with a conserved domain present in lap1 and a novel er protein
-site specific intrabodies to decrease and prevent generation of alzheimer's a peptide
trpv4 channel is involved in the coupling of fluid viscosity changes to epithelial ciliary activity
chromatin decondensation in s-phase involves recruitment of cdk2 by cdc45 and histone h1 phosphorylation
activity-dependent and -independent nuclear fluxes of hdac4 mediated by different kinases in adult skeletal muscle
the high mobility group transcription factor sox8 is a negative regulator of osteoblast differentiation
inhibition of ngf deprivation
akt2 phosphorylates synip to regulate docking and fusion of glut4-containing vesicles
two modes of exocytosis at hippocampal synapses revealed by rate of fm1-43 efflux from individual vesicles
rock-i regulates closure of the eyelids and ventral body wall by inducing assembly of actomyosin bundles
raf-1 regulates rho signaling and cell migration
a small surface hydrophobic stripe in the coiled-coil domain of type i keratins mediates tetramer stability
golgi positioning : are we looking at the right map
the yeast s phase checkpoint enables replicating chromosomes to bi-orient and restrain spindle extension during s phase distress
hrp59, an hnrnp m protein in chironomus and drosophila, binds to exonic splicing enhancers and is required for expression of a subset of mrnas
a systems analysis of importin-
targeting of arf-1 to the early golgi by membrin, an er-golgi snare
arfgap1 dynamics and its role in copi coat assembly on golgi membranes of living cells
sox2 induction by fgf and fgfr2 activating mutations inhibits wnt signaling and osteoblast differentiation
tgf-1 activates two distinct type i receptors in neurons : implications for neuronal nf-b signaling
1 integrin and il-3r coordinately regulate stat5 activation and anchorage-dependent proliferation
the extracellular domains of fasl and fas are sufficient for the formation of supramolecular fasl-fas clusters of high stability

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